The unique physiology of solid tumors: opportunities (and problems) for cancer therapy. Cancer Res 1998 Apr 1;58(7):1408-1416.
Effect on tumor cells of blocking survival response to glucose deprivation. J Natl Cancer Inst 2004 Sep 1;96(17):1300-1310.
Mol Cell 2002 Nov;10(5):983-994.
Roles of CHOP/GADD153 in endoplasmic reticulum stress. Cell Death Differ 2004 Apr;11(4):381-389.
Borrelidin induces the unfolded protein response in oral cancer cells and chop-dependent apoptosis. ACS Med Chem Lett 2015 Sep 8;6(11):1122-1127.
Decreased GRP78 protein expression is a potential prognostic marker of oral squamous cell carcinoma in Taiwan. J Formos Med Assoc 2010 May;109(5):326-337.
CHOP is a multifunctional transcription factor in the ER stress response. J Biochem 2012 Mar;151(3):217-219.
The critical role of JNK in the ERmitochondrial crosstalk during apoptotic cell death. J Cell Physiol 2012 May;227(5):1791-1795.
JNK1/2 expression and modulation of STAT3 signaling in oral cancer. Oncol Lett 2016 Jul;12(1):699-706.
Proteasome inhibitor PS-341 induces apoptosis through induction of endoplasmic reticulum stress-reactive oxygen species in head and neck squamous cell carcinoma cells. Mol Cell Biol 2004 Nov;24(22):9695-9704.
A new classification for potentially malignant disorders of the oral cavity. Oral Oncol 2011 Sep;47(9):920-923.
Oral potentially malignant disorders: a proposal for terminology and definition with review of literature. J Oral Maxillofac Pathol 2014 Sep;18(Suppl 1):S77-S80.
